tonsurans, other anthropophilic species, such as T violaceum, have appeared and in- creased in prevalence in urban populations in the UK and Europe.
The second factor is changes in immigration patterns and the increase in international travel which might have facilitated the spread of T. tonsurans, which is less susceptible to griseofulvin, to become predominant. audouinii is susceptible to this antifungal agent, this species may have been eradicated, allowing T. The first is the wide- spread use of griseofulvin to treat scalp infections. At least two factors are thought to be responsible for this shift in species distribution. The predominant aetiological agents of anthropophilic tinea capitis differ from one region to another, and can change within a particular region over time. The disease seldom persists beyond the age of 16. It is primarily a disease of pre-pubertal children, being more common in males than females, and most prevalent between 6 and 10 years of age. Tinea capitis caused by anthropophilic Microsporum and Trichophyton species is a contagious disease endemic in many countries. It occurs mainly in prepubescent children. Tenia capitis has been distributed world-wide, but is more common in Africa, Asia and southern and eastern Europe. Geographical distribution of Tinea capitis:. Certain individuals are termed as carriers, as dermatophytes can be isolated from their scalp but they lack any signs and symptoms. The major clinical signs are hair loss and scaling along with the possibilities of inflammation. The term tinea capitis is used to refer the infections caused by the dermatophytes on the hair scalp and hair skin. Tinea capitis: etiology, clinical manifestation, diagnosis and treatment What is Tinea capitis?
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